For the emergence of unstable walking, I believe that this is a problem that most elderly people will encounter. When people get older, their legs are not very flexible, and it is normal for them to walk unsteadily. Therefore, the unstable walking of the elderly may be the following three situations.
1¡¢ Lack of vitamin B12, vitamin B12, will not only cause anemia, but also cause spinal cord diseases such as spinal deformity, resulting in weakness of both lower limbs, walking instability and other symptoms.
2¡¢ Potassium deficiency, hypokalemia more prominent performance, is the limbs weakness, lower limbs more obvious, beans, vegetables, fruits eat less, summer sweating is easy to cause potassium deficiency.
3¡¢ Cerebral infarction, many people think that cerebral infarction will appear hemiplegia, hands and feet are not flexible, in fact, walking instability, often dizziness is also the early symptoms of cerebral infarction.
In life, often walk unsteadily, refers to walk crooked, tottering appearance, is very laborious appearance.
There are many reasons for unstable walking, simple excessive fatigue, fever, cervical spondylosis, illness, body deficiency, hypoglycemia can appear when walking instability, generally can be relieved in a short time. If a long time of dizziness, strong vertigo, accompanied by walking instability, we need to consider the following situations: 1. Peripheral vestibular lesions: benign paroxysmal positional vertigo, labyrinthitis, Meniere’s disease caused by organ degeneration of the inner ear, which are often accompanied by vertigo, evil heart and vomiting. 2¡¢ Cerebral vascular disease, especially in the cerebellum and brainstem, can lead to vertigo, nausea, vomiting and balance disorders for a long time. Cerebellar hemorrhage can also appear vertigo, walking instability symptoms, this situation needs to be treated in a regular hospital in time.
There are three main clinical cases of unstable walking.
The first is cerebellar lesions, because the cerebellum is an important balance of our body. Cerebellar lesions usually lead to walking broad basal posture. The degree is that the two legs are separated very wide when walking. This is to make the body’s center of gravity down and prevent falling down. Cerebellar lesions can be examined by MRI of the brain.
Second, vestibular dysfunction can also lead to walking instability, we call vestibular ataxia, this MRI can also find vestibular lesions.
Third, is the feeling barrier, mainly is the deep feeling barrier, this kind of situation when walking unsteadily will have the foot to step on the cotton feeling, the feeling foot walks is not steadfast.
There are many reasons for unstable walking, which can be caused by central nervous system diseases or peripheral diseases. Peripheral degeneration, including toxic metabolic peripheral neuropathy, multiple nerve root neuritis, tuberculosis, etc. Brain lesions, especially cerebellar lesions, can cause cerebellar ataxia, lead to walking instability, frontal lobe tumor, ischemic cerebrovascular disease and so on can cause walking instability.
Unstable walking may be a kind of balance disorder. Balance training can be carried out. At the same time, we should analyze the cause of the disease and treat it accordingly. The common lesions in the cerebellum, including cerebrovascular diseases, are mainly seen in cerebellar infarction and cerebellar hemorrhage, and may be caused by cerebellar tumors or inflammation. If it is cerebrovascular disease, such as cerebral hemorrhage patients, early suitable for surgery, surgical treatment can be considered. If it is cerebral infarction, ultra early suitable for thrombolysis, intravenous thrombolytic therapy, and can be given neuroprotective treatment, and if necessary, antihypertensive treatment. The commonly used drugs are mannitol and glycerol fructose. If it is caused by cerebellar tumor, surgery or radiotherapy can be performed. If it is a benign tumor, the symptoms may be relieved after surgery, and the treatment effect of patients with malignant tumor is poor. If it is caused by inflammation, it mainly includes viral infection, or bacterial infection, mainly antiviral treatment, or anti-inflammatory treatment. Most patients can be cured by active and correct treatment.
It may be too tired, or there may be something wrong with the cerebellum. The function of the cerebellum is: through its rich afferent and efferent connections with the brain, brainstem and spinal cord, the cerebellum participates in the regulation of body balance and muscle tension (muscle tension), as well as the coordination of random movement. The cerebellum is like a big regulator. When people are drunk, they will walk unsteadily because alcohol paralyzes the cerebellum. In an experiment, when a dog is removed from its cerebellum, the dog will lose its coordination when walking. The regulation of the cerebellum for body balance is carried out by the flocculus nodule, that is, the ancient cerebellum. The balance regulation of the body is a reflex process, and the flocculus nodule is the central device of this reflex activity. The information of the change of body balance is sensed by vestibular organs. It is transmitted to the chorion nodule of cerebellum through vestibular nerve and vestibular nucleus. According to this, the cerebellum sends out the regulating impulse to the body balance. It reaches the motor neurons of the anterior horn of the spinal cord through the vestibular spinal tract, and then to the muscles through the spinal nerve, so as to coordinate the movement and tension of the antagonistic muscle groups, so as to keep the body in balance. For example, when a person is standing and his head is back, the knee and ankle joints will automatically flex to resist the transfer of body center of gravity caused by head backward, so that the body can keep balance without falling. In this process, the auxiliary flexion of the knee and ankle joint for head backward is the result of the regulatory impulse from the cerebellum to coordinate the movement and tension of the muscles concerned. If the flocculus nodule is injured, the balance function of the body will be destroyed. Monkeys who have removed the floccule nodule can not stand. They always sit in the corner of the cage, supporting the body on both sides of the cage to maintain balance. In humans, if the flocculus is damaged or oppressed, the patient’s body balance will be seriously out of balance, the body will be tilted, and the gait will be staggered when walking. The study also showed that the vermis cortex also received the proprioception and visual impulse related to body balance, and there were many fibers between the parietal nucleus and vestibular nucleus. Therefore, the longitudinal medial area, which is composed of vermis cortex and parietal nucleus, is also involved in the regulation of body balance, mainly standing. Injury to the medial area will also cause difficulty in balancing and standing. The regulation of tensor is a basic reflex activity in which different muscle fiber groups of muscles contract in turn, so that the whole muscle is in a state of frequent mild contraction, thus maintaining the body standing posture as a basic reflex activity. Cerebellum can regulate muscle tension activity, and its regulatory effect is manifested in two aspects: inhibition of muscle tension and facilitation of muscle tension. The inhibitory effect of cerebellum on muscle tension is mainly the function of vermis in the anterior lobe (old cerebellum), which is most obvious in the brain removed animals. Stimulation of the vermis in the anterior cerebellar lobe of a decapitated cat can reduce the excessive tension of the extensor muscles caused by the removal of the brain; on the contrary, the removal of this part will strengthen the rigidity of the denervated brain. The facilitation of cerebellum to muscle tension is realized by the two sides of anterior lobe. Stimulation of both sides of the anterior cerebellar lobe in monkeys can strengthen the tension of extensor muscles and reduce the tension of laminar muscles; in humans, the injury of this part can cause myasthenia or hypotensivity. The inhibition or facilitation of anterior cerebellar lobe on muscle tension is realized through the inhibition and facilitation regions of muscle tension in the reticular structure of brain stem. These two regions are the central areas controlling skeletal muscle tension. They control the activity of gamma motor neurons in the anterior horn of the spinal cord through the descending reticular spinal tract. The descending impulse of facilitation area can strengthen the activity of ¦Ã – motoneurons and strengthen the muscle tension; the inhibitory area can weaken the activity of ¦Ã – motoneurons and weaken the muscle tension. Under normal circumstances, the activities of the inhibition and facilitation areas of the brainstem reticular structure maintain a dynamic balance under the influence of the high-level central brain, striatum and cerebellum, so as to maintain the muscle tension in a normal state. If for some reason, the cerebellum (the vermis or the lateral part of the anterior lobe) strengthens or weakens the activity of the inhibition or facilitation area of the muscle tension in the reticular structure of the brain stem The effect will destroy the balance between the two low-level centers and strengthen or weaken the muscle tension activity. In addition, the cerebellum can also regulate muscle tension through the lateral vestibular nucleus. From the lateral vestibular nucleus, the vestibular spinal tract reaches the spinal cord. Through this descending conduction tract, the tension impulse increases the activity of ¦Á – motoneurons in the anterior horn of the spinal cord and strengthens the muscle tension. There are direct and trans parietal indirect projections from vermis cortex to lateral vestibular nucleus. The direct fiber projection is an inhibitory pathway for the lateral vestibular nucleus. It reduces the activity of ¦Á – motor neurons in the anterior horn of the spinal cord, resulting in the weakening of muscle tension The indirect projection to the lateral vestibular nucleus is an excitatory pathway through which the parietal nucleus can enhance the activity of the lateral vestibular nucleus, and the ultimate result is the enhancement of muscle tonic activity. Therefore, the damage to the vermis cortex strengthened the rigidity of the denervated animals, while the injury of the parietal nucleus weakened the muscle tension of the decapitated animals. The coordination of random movement is the intentional movement initiated by the cerebral cortex, and the coordination of random movement is completed by the hemispheric part of the cerebellum, namely the new cerebellum. The damage of the new cerebellum will make the victim’s muscle tension drop and the coordination disorder of random movement, which is called cerebellar ataxia. The main manifestations are as follows: ¢Ù the accuracy of movement is impaired. When the patient pays attention to do a certain action, such as using the finger nose, the finger trembles. The closer to the target, the more severe the finger trembles. Therefore, the accurate direction of movement cannot be grasped. ¢Ú The coordination of movements was impaired. Patients lose the ability to stop a movement and immediately switch to the opposite direction of action, and the movement decomposition is discontinuous. For example, if the patient can not complete the simple and fast rotation of the palm, which is called rotation disorder; when completing one direction of movement and need to change the direction of movement, the patient must stop to think about the next step before starting a new movement. All the listed symptoms were only found in motor activity, indicating that the new cerebellum plays an important coordinating role in voluntary movement, which is the result of the regulation of the cerebellum on the activities of cerebral cortex and spinal cord. There is a two-way neural connection between the cerebral cortex and the cerebellum. In the descending process, the pyramidal tract, which transmits motor information, has branches in the pontine nucleus to replace neurons, and then the pontine nucleus sends out fibers into the cerebellum to form the cortex pontine cerebellum tract. The projection from the cerebellum to the cerebral cortex is projected from the axons of Purkinje cells in the neocerebellar cortex From the dentate nucleus, fibers are sent out from the dentate nucleus to the motor area of the cerebral cortex through the ventrolateral nucleus of the thalamus. This is the dentate nucleus thalamic cortical tract. These two conduction bundles constitute the basic circuit of the cerebellum in regulating the activities of the motor areas of the cerebral cortex. When the motor areas of the cerebral cortex transmit the motor impulses that cause muscle contraction to the spinal cord through the pyramidal tract, there are also collateral impulses that reach the cerebellum through the cortical pontine cerebellar tract. When the related muscles contract after receiving these motor impulses, proprioceptive receptors such as muscle spindles in muscles transmit the proprioceptive impulses related to muscle movements to the cerebellum through the spinocerebellar tract. In this way, at every moment of random movement, the cerebellum not only receives the command from the cerebral cortex, but also obtains the information of the muscle executing the movement instruction. After comparing the two, the impulses from Purkinje cells in cerebellar cortex adjust the activities of deep cerebellar nuclei, mainly dentate nucleus, and then the impulses from dentate nucleus thalamic cortex bundle feed back to the motor areas of cerebral cortex, and adjust the activities of motor areas of cerebral cortex by facilitation or inhibition. On the other hand, the cerebellum receives the proprioceptive information of muscle movement from the spinocerebellar tract, and transmits regulatory impulses to the spinal cord through the red nucleus and the red nucleus spinal tract to regulate the activity of motor neurons. The cerebellum is just like this. In the process of random movement, the cerebellum immediately and continuously adjusts the activities of the motor area of cerebral cortex, red nucleus and spinal cord, so that the movement can be carried out accurately, smoothly and smoothly. The lateral and medial parts of neocerebellar cortex and their corresponding projection nuclei, dentate nucleus and interposition nucleus, play different roles in the initiation and completion of random movement. The lateral area and dentate nucleus of the cerebellar cortex, through the interaction between them and the cerebral cortex, participated in the design and programming of random movement together with the cerebral cortex contact area, basal ganglia, ventrolateral thalamic nucleus and other neural structures in the early stage of voluntary movement; while the interpositional area and interpositional nucleus of the cerebellar cortex participated in the execution of random movement. For example, before the monkey began to do wrist flexion or extension, there were changes in the discharge of dentate nucleus and interpositional nucleus in the deep cerebellum. However, the discharge changes of dentate nucleus cells occurred in front of interpositional nucleus cells, and the discharge pattern was more complex than that of interpositional nucleus cells. The difference of reaction time and reaction pattern showed that the two longitudinal areas of cerebellum hemisphere and their The corresponding projective nuclei play different roles in random motion. In addition, the cerebellum is also related to motor learning and memory and cardiovascular activities. During the formation and maintenance of niching conditioned reflex in rabbits, the relevant neurons of hippocampal CA1, CA3, hemispheric part of the sixth lobule of cerebellar cortex (H VI) and intermedian nucleus can produce learning related discharges. Damage of cerebellar cortex h ¢ö and intermedian nucleus can make the above conditioned reflex and the Learning Association of hippocampal CA1 and CA3 disappear. Electrical stimulation of the rostral part of the cerebellar fastigial nucleus can cause obvious cardiovascular reactions, including the obvious increase of arterial blood pressure, increased heart rate, abnormal rhythm, changes in pressure sensitivity and chemosensitivity modulation, etc. this cardiovascular response is called the parietal nucleus pressor response.
If there is any problem, go to the hospital for examination.
If you are walking normally, you suddenly feel unstable and dizzy. Although it may not sound good, it is very likely that there is something wrong with your body. It is suggested that you go to the hospital for a comprehensive examination, and health is the most important thing.
If you squat for a long time or sit for a long time and suddenly get up, the whole body’s blood can’t be perfect in time. As a result, the brain will lack oxygen, which will cause dizziness and unstable walking. This is a normal reaction. Don’t worry about it.
There are several reasons why you can walk unsteadily! First, I don’t know how old you are. If you are older, it may be caused by insufficient cerebral blood supply, and high or low blood pressure may lead to unstable walking! In addition, it is possible to walk fast and lead to instability. It is also possible that the foot is not stable! It is suggested that you should often go to the hospital to have a check-up to see what the situation is. Health is the most important thing. Don’t neglect it. If you go to the hospital to have a look, it will be OK. Don’t delay it!